Bebe Babbitt
Kathleen Parks
Perry Solomon
26 y/o
5′ 6″ (168 cm)
122.0 lb (55.5 kg)
Reason for encounter
More frequent severe headaches
Location
Outpatient clinic with x-ray, ECG, and laboratory capabilities
History Questions
- How can I help you today?
- Do you have any other symptoms or concerns we should discuss?
- Tell me how does that makes you feel?
- How does this affect your life?
- Is there any thing I can do for you right now?
- Do you have a headache?
- When did your headache start?
- How long does your headache last?
- Does your headache come and go?
- Have you been having fevers?
- ……….
Physical Exams Required
- Weight
- Height
- cognitive status
- SpO2
- temperature
- blood pressure
- pulse
- respiration
- assess stance & gait
- …………….
Get all the complete Bebe Babbitt Kathleen Parks case with history questions, physical exam, complete EHR documentation, exercises, case findings, diagnosis, test results, and management plan.
Diagnosis Feedback
Migraine with aura
Migraine is said to occur as a result of primary neuronal dysfunction. Cortical spreading depression (a wave of depolarization of glia and neurons that makes its way across the cortex and is self-propagating) is associated with migraine aura and headache. This wave of depolarization activates trigeminal afferent neurons. The trigeminal afferent neurons innervate blood vessels in cerebral parenchyma, pia, dura mater, and venous sinuses, thus activating the trigeminovascular system. Once the trigeminal ganglion is activated, vasoactive peptides like substance P and calcitonin gene-related peptides are released, leading to inflammation. Calcitonin gene-related peptide is a potent vasodilator. Hence, as a part of the inflammatory process, vasodilation occurs. Inflammation then leads to a sensitization of neurons, which become increasingly sensitive to nociceptive stimuli.
